{"id":2240,"date":"2013-04-11T16:45:00","date_gmt":"2013-04-11T20:45:00","guid":{"rendered":"https:\/\/med.sites.unc.edu\/wolberglab\/images\/arterial-thrombosis\/"},"modified":"2018-04-10T14:01:28","modified_gmt":"2018-04-10T18:01:28","slug":"arterial-thrombosis","status":"publish","type":"page","link":"https:\/\/www.med.unc.edu\/wolberglab\/arterial-thrombosis\/","title":{"rendered":"Arterial Thrombosis"},"content":{"rendered":"<div>\n<p><span style=\"color: #666666; letter-spacing: normal; line-height: 22.875px; text-align: start; text-indent: 0px; text-transform: none; word-spacing: 0px; text-size-adjust: auto; text-stroke-width: 0px; background-color: #ffffff; display: inline !important; float: none;\"><b>Interplay between abnormalities in blood components, the vasculature, and blood flow contribute to the development of arterial thrombosis.<\/b> Arterial thrombosis involves the formation of platelet-rich \u201cwhite clots\u201d that form after rupture of atherosclerotic plaques and exposure of procoagulant material such as lipid-rich macrophages (foam cells), collagen, tissue factor, and\/or endothelial breach, in a high shear environment. TM = thrombomodulin; II = prothrombin; IIa = thrombin; Fgn = fibrinogen; TF = tissue factor.<\/span><\/p>\n<p><i>Wolberg AS, Aleman MM, Thrombosis Research 2010 Apr;125 Suppl 1:S35-7<\/i><\/p>\n<p><i><img decoding=\"async\" class=\"image-inline\" title=\"Arterial thrombosis\" src=\"https:\/\/www.med.unc.edu\/wolberglab\/wp-content\/uploads\/sites\/645\/2018\/04\/arterial-thrombosis.jpg\" alt=\"Interplay between abnormalities in blood components, the vasculature, and blood flow contribute to the development of arterial thrombosis. Arterial thrombosis involves the formation of platelet-rich \u201cwhite clots\u201d that form after rupture of atherosclerotic plaques and exposure of procoagulant material such as lipid-rich macrophages (foam cells), collagen, tissue factor, and\/or endothelial breach, in a high shear environment. TM = thrombomodulin; II = prothrombin; IIa = thrombin; Fgn = fibrinogen; TF = tissue factor.\" \/><br \/>\n<\/i><\/p>\n<\/div>\n","protected":false},"excerpt":{"rendered":"<p>Interplay between abnormalities in blood components, the vasculature, and blood flow contribute to the development of arterial thrombosis. Arterial thrombosis involves the formation of platelet-rich \u201cwhite clots\u201d that form after rupture of atherosclerotic plaques and exposure of procoagulant material such as lipid-rich macrophages (foam cells), collagen, tissue factor, and\/or endothelial breach, in a high shear &hellip; <a href=\"https:\/\/www.med.unc.edu\/wolberglab\/arterial-thrombosis\/\" aria-label=\"Read more about Arterial Thrombosis\">Read more<\/a><\/p>\n","protected":false},"author":9746,"featured_media":0,"parent":0,"menu_order":30,"comment_status":"closed","ping_status":"closed","template":"","meta":{"_acf_changed":false,"footnotes":"","_links_to":"","_links_to_target":""},"class_list":["post-2240","page","type-page","status-publish","hentry","odd"],"acf":[],"_links_to":[],"_links_to_target":[],"_links":{"self":[{"href":"https:\/\/www.med.unc.edu\/wolberglab\/wp-json\/wp\/v2\/pages\/2240","targetHints":{"allow":["GET"]}}],"collection":[{"href":"https:\/\/www.med.unc.edu\/wolberglab\/wp-json\/wp\/v2\/pages"}],"about":[{"href":"https:\/\/www.med.unc.edu\/wolberglab\/wp-json\/wp\/v2\/types\/page"}],"author":[{"embeddable":true,"href":"https:\/\/www.med.unc.edu\/wolberglab\/wp-json\/wp\/v2\/users\/9746"}],"replies":[{"embeddable":true,"href":"https:\/\/www.med.unc.edu\/wolberglab\/wp-json\/wp\/v2\/comments?post=2240"}],"version-history":[{"count":0,"href":"https:\/\/www.med.unc.edu\/wolberglab\/wp-json\/wp\/v2\/pages\/2240\/revisions"}],"wp:attachment":[{"href":"https:\/\/www.med.unc.edu\/wolberglab\/wp-json\/wp\/v2\/media?parent=2240"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}