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Jin SzatkiewiczDepartment of Genetics
Diana PerkinsDepartment of Psychiatry
Weili LinDepartment of Radiology
Khalilah JohnsonDepartment of Allied Health Sciences
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Clare HarropDepartment of Allied Health Sciences
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Dara ChanDepartment of Allied Health Sciences
Brian BoydSchool of Education
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Jonathan BergDepartment of Genetics
Marissa DiPieroDr. Marissa DiPiero (2nd year T32 awardee) received her PhD in Neuroscience from the University of Wisconsin-Madison under the mentorship of Dr. Doug Dean. Her research focuses on the development and utilization of diffusion MRI methodologies to study brain microstructure across the lifespan and uncover differences in brain-behavior relationships associated with autism. As a UNC CIDD T32 fellow, Dr. DiPiero works with Dr. John Gilmore to investigate individual differences in the developmental trajectories of brain-behavior relationships in children at an increased risk for neurodevelopmental conditions, including autism and schizophrenia.
Scott AlbertDr. Scott Albert (2nd year T32 awardee) received his PhD in Biomedical Engineering from Johns Hopkins University working with Dr. Reza Shadmehr. His research examines resilience in motor circuits across humans and mice. As a UNC CIDD T32 Fellow, Dr. Albert works with Dr. Adam Hantman, in examining whole-brain maps of motor activity in mouse models of Angelman Syndrome and Dup15q Syndrome. He will use fMRI and electrophysiology to identify how UBE3A expression regulates the adaptability and robustness of motor control circuits.
Samuel BarthDr. Samuel Barth (2nd year T32 awardee) received his PhD in Neuroscience from Wake Forest University working with Dr. Kimberly Raab-Graham. His research examines how mTOR dysregulation affects GABAergic synaptic formation and elimination in a neurodevelopmental disorder called Tuberous Sclerosis Complex (TSC). As a UNC CIDD T32 Fellow, Dr. Barth examines the molecular contributions to Dup15q and Angelman syndromes while working with Dr. Ben Philpot. His research will disambiguate how differential expression of Ube3A contributes to Dup15q and Angelman syndrome pathology.