Their research, published in the International Journal of Molecular Sciences, indicates T and B lymphocyte transcriptional states differentiate between sensitized and unsensitized patients affected by alpha-gal syndrome (AGS).
Further research into AGS is needed, because the mechanisms of pathogenesis driving the syndrome are not fully understood. Differences in immune gene expression between AGS individuals and non-allergic controls may illuminate molecular pathways and targets critical for AGS development.
You can read the recently published paper via this link.